Read e-book online Anaesthesia, Pain, Intensive Care and Emergency A.P.I.C.E.: PDF

By K. Hillman (auth.), Antonino Gullo M.D. (eds.)

ISBN-10: 8847005701

ISBN-13: 9788847005709

ISBN-10: 884700571X

ISBN-13: 9788847005716

Developing sectors within the extensive Care box – and in serious Care drugs ordinarily – require particular degrees of competence having a related universal denominator: an in-depth wisdom of human pathophysiology. even though this quantity offers lots of themes in consistent evolution, as witnessed through the gathering of chapters compiled through a number of researchers, this version comprises, specifically, fields within which decision-making on the patient’s bedside prevails over theoretical argumentation. In different phrases, the 1st and leading message this variation desires to supply is for the reader to concentration his/her cognizance on evidence-based medicine.

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Read or Download Anaesthesia, Pain, Intensive Care and Emergency A.P.I.C.E.: Proceedings of the 21st Postgraduate Course in Critical Care Medicine Venice-Mestre, Italy — November 10–13, 2006 PDF

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Extra resources for Anaesthesia, Pain, Intensive Care and Emergency A.P.I.C.E.: Proceedings of the 21st Postgraduate Course in Critical Care Medicine Venice-Mestre, Italy — November 10–13, 2006

Example text

West JB, Mathieu-Costello O (1995) Stress failure of pulmonary capillaries as a limiting factor for maximal exercise. Eur J Appl Physiol 70:99–108 22. Parker JC, Ivey CL, Tucker JA (1998) Gandolinium prevents high airway pressure-induced permeability increases in isolated rat lungs. J Appl Physiol 84:1113–1118 23. Robertson B (1984) Pulmonary surfactant. Elsevier, Amsterdam 24. Muscedere JG, Mullen JB, Gun K, Slutsky AS (1994) Tidal ventilation at low airway pressures can augment lung injury. Am J Respir Crit Care Med 149:1327–1334 25.

14 R. J. Haitsma, H. Zhang ciated ligands [11]. For example, TLR4 is crucial for lipopolysaccharide (LPS) recognition and transduces its signal across the cell membrane [12]. Based on their cytoplasmic domains, mammalian TLRs are also homologous to members of the interleukin-1 receptor (IL-1R) family. MyD88 is an adaptor protein that associates with the IL-1R protein by direct TLR–TLR domain interaction [13]. The death domain (DD) of MyD88, then, is recruited by a homotypic DD interaction, a family of IL-1R-associated protein kinases (IRAKs) [13, 14].

Pittet et al. demonstrated that mice lacking the integrin vb6 (activating TGF-b) were completely protected from pulmonary oedema in a model of bleomycin-induced ALI [24]. Inhibition of TGF-b (pharmacologically) also protected wild-type mice from pulmonary oedema induced by bleomycin or E. Thus, integrin-mediatedlocalactivation of TGF-b is critical to the development of pulmonary oedema in ALI, and blocking TGF-b or its activation could be a possible treatment for ALI. 16 R. J. Haitsma, H. Zhang ACE-2 in ALI The renin–angiotensin system has an important role in the maintenance of both blood pressure homeostasis and fluid and salt balance.

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Anaesthesia, Pain, Intensive Care and Emergency A.P.I.C.E.: Proceedings of the 21st Postgraduate Course in Critical Care Medicine Venice-Mestre, Italy — November 10–13, 2006 by K. Hillman (auth.), Antonino Gullo M.D. (eds.)


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