Awareness of Deficit after Brain Injury: Clinical and by Daniel L. Schacter, George P. Prigatano PDF

By Daniel L. Schacter, George P. Prigatano

ISBN-10: 0195059417

ISBN-13: 9780195059410

This quantity presents, for the 1st time, multidisciplinary views at the challenge of wisdom of deficits following mind harm. Such deficits may well contain belief, consciousness, reminiscence, language, or motor features, and so they can heavily disrupt an individual's skill to operate. besides the fact that, a few brain-damaged sufferers are totally blind to the lifestyles or severity in their deficits, even if they're simply spotted through others. In addressing those issues, members disguise the total diversity of neuropsychological syndromes within which issues of information of deficit are saw: hemiplegia and hemianopia, amnesia, aphasia, demanding head damage, dementia, and others. at the scientific aspect, prime researchers delineate the consequences of knowledge of deficits for rehabilitation and sufferer administration, and the position of safeguard mechanisms akin to denial. Theoretical discussions specialize in the significance of knowledge disturbances for higher figuring out such cognitive tactics as cognizance, attention, and tracking.

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Extra resources for Awareness of Deficit after Brain Injury: Clinical and Theoretical Issues

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The positive correlation between anosognosia related to motor impairment and unilateral neglect, both personal and extrapersonal, was evident. Instances of double dissociation were found, however. As expected, they were more frequent with extrapersonal than with personal neglect. Denial of paralysis in the absence of neglect of the paralyzed limb is theoretically important (see next section). No significant correlation was found between unawareness of hemianopia and sensorimotor disorders. This finding is far from surprising and does not seem to have any theoretical implications.

In this case, the output of the representational network (layer IV) faithfully mirrors outside reality. Two kinds of lesions of the model may be envisaged with reference to anosognosia and, more generally, unilateral misrepresentation including hemineglect. In one case (Fig. 2-2), one side of the topologically organized representational network is completely destroyed or at least inactivated. In this case, only 28 AWARENESS OF DEFICIT AFTER BRAIN INJURY Figure 2-2. Dysfunction underlying hemineglect.

Visuo-somatosensory) that, especially in the case of unawareness of hemiplegia, may complicate the clinical picture. The model largely ignores the problem of its actual implementation in the two cerebral hemispheres, including the problem of left/right asymmetries, although a suggestion was made by Bisiach and Vallar (1988). The afferent component of the model (Fig. 2-1, layer I) is a sensory transducer transmitting information relative to that portion of the stimulus array that at any given instant falls within the field of a sensory receptor.

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Awareness of Deficit after Brain Injury: Clinical and Theoretical Issues by Daniel L. Schacter, George P. Prigatano


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